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While the exact mechanism of release is a matter of debate, several molecules have been implicated in regulating long-term plasticity including D-serine, glutamate and ATP/adenosine (see Figure 2 for a schematic of pathways)

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Contrary to our initial prediction of a telomere reduction among BSO-treated birds, the delayed effect of the early BSO treatment on females’ telomere length suggests a compensatory (hormetic) response to early oxidative stress, matching some of our previous findings in females in the same captive population (Romero-Haro and Alonso-Alvarez 2015)